In this project, we are testing the hypothesis that chronic intermittent hypoxia exacerbates WTC dust exposure induced oxidative stress, lung injury, and alterations in lung function in a mouse model of WTC dust exposure. We will also examine alterations in the metabolomic profile and correlate them to the changes in the immunometabolic function of alveolar and interstitial lung macrophages in this mouse model of WTC dust exposure.
No publications available at this time.
